Patent · US Expired

Mice and cells with a homozygous disruption in the RNase L gene and methods therefore

US6028243A · kind A · utility

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14Claims
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Assignee

Inventors

Key dates

Filing dateOct 3, 1997
Grant dateFeb 22, 2000
Priority date
Expiry dateOct 3, 2017

Classification

  • Technology area (CPC C)Chemistry; Metallurgy
  • CPC primaryC12N2517/02
  • WIPO fieldBiotechnology
  • WIPO sectorChemistry

Abstract

The present invention provides a mutant, non-human mammal, particularly a mutant mouse, having a homozygous disruption in the RNase L gene thereof. Since the homozygous disruption in the RNase L gene leads to minimal if any production of RNase L in the mutant mammals, such mutant mammals are useful for assessing the effect of antiviral drugs on the induction, synthesis, or activation of RNase L. The present invention also relates to mutant, non-human, embryonic stem cell lines having a heterozygous disruption of the RNase L gene thereof, to isolated mammalian cells having a homozygous disruption in the RNase L gene thereof, and to a DNA construct comprising a DNA sequence of a disrupted coding exon of a RNase L gene.

Source: USPTO / EPO open patent data. Objective bibliographic and citation counts.